How Much Vitamin D Do You Really Need to Take? Last November 3, 2. University of Toronto, Ontario, Canada, noted doctors Cedric Garland and Tracey O'Connor ran a seminar on how vitamin D can be used to prevent breast cancer - - as well as infectious diseases, type 1 diabetes, hypertension, colon cancer, and falls in the elderly. Presenters included some of the best known vitamin D researchers/practitioners, such as Robert P. Heaney, Reinhold Vieth, John White, and Susan Whiting. It is estimated that 2. D serum levels. The conference looked at the current research and practice with vitamin D to enable everyone to take action today based on what's known to solve the deficiency epidemic, and to start the prevention of many diseases. Vitamin D Dose Recommendations. Below 5. 35 units per pound per day. Age 5 - 1. 02. 50. Adults. 40. 00- 8. Pregnant Women. 50. WARNING. There is no way to know if the above recommendations are correct. The ONLY way to know is to test your blood. You might need four to five times the amount recommended above. Ideally, your blood level of 2. OH)D should be 6. What is the treatment for a vitamin D deficiency? Consuming high amounts of vitamin A can result in hair loss. The tolerable upper limit of the vitamin for those over age 19 is. As more and more scientific evidence emerges, confirming that currently recommended daily allowances (RDA) of vitamin D are grossly insufficient for young and old alike, many have asked me to clarify the recommended dosages, especially as it pertains to children. General Information About Adult Vitamin D Requirements Before I begin, I want to emphasize that under summer conditions it is frequently possible to generate about 2. D by exposing your skin to the sun. NEW, smaller softgels! High-potency vitamin D supplement supplies 5,000 IU per single . Supports maintenance of healthy bones and teeth. If you weigh more than the average person your age your vitamin d requirements will need to be adjusted upward; Vitamin D levels tend to be low in obese individuals. That fact makes these recommendations seem more in line with reality. Currently, the U. S. RDA for vitamin D is 4. IUs (international units) for the majority of the population. Interestingly, the majority of people I see in my travels that are taking vitamin D are taking 1,0. Don’t fool yourself, as an adult, you likely need about 8,0. IUs a day. Some also worry that if they are in the sun that they will overdose on vitamin D. Although most people take vitamin D supplements without any problems, it. This is called vitamin D toxicity. Vitamin D toxicity, where.However this is not typically the case, and here’s why: When you’re exposed to the sun, the UVB rays cause vitamin D to be produced in your skin while the UVA rays in the sunlight will tend to destroy excessive levels of vitamin D circulating in your body. It is somewhat of a natural failsafe mechanism that prevents overdosing. HOWEVER, please understand that about 1. I have seen people requiring over 3. D a day to reach therapeutic levels of 2. D in their blood. Please remember that the ONLY way to know for sure is to get your blood level tested, which I’ll go over in just a moment. Current RDA Guidelines for Vitamin D Are Outdated in Light of New Research. At the end of 2. 00. American Academy of Pediatrics doubled its recommended dose of vitamin D for infants, children, and adolescents, raising it from 2. Unfortunately, this is still a woefully inadequate recommendation for children. Research reveals children may need ten times that amount in order to receive the health benefits that optimal vitamin D levels have to offer. As of right now, the conventional RDAs are only: 4. IUs for infants, children and adolescents. IUs for adults up to age 5. IUs for adults aged 5. IUs for seniors over 7. Recommended Daily Intake for Optimal Health. Based on the most recent research, the current recommendation is 3. IUs of vitamin D per pound of body weight. So for a child weighing 4. IUs daily, and for a 1. IUs. However, it’s important to realize that vitamin D requirements are highly individual, as your vitamin D status is dependent on numerous factors, such as the color of your skin, your location, and how much sunshine you’re exposed to on a regular basis. So, although these recommendations may put you closer to the ballpark of what most people likely need, it is simply impossible to make a blanket recommendation that will cover everyone’s needs. So how do you ensure optimal vitamin D levels for yourself, your child, and aging parents? Blood Testing Is the ONLY Reliable Way to Determine How Much Vitamin D You or Your Child Needs. Yes, the only way to determine the correct dose is to get your blood tested since there are so many variables that influence your vitamin D status. I recommend using Lab Corp in the U. S. This is the marker that is most strongly associated with overall health. Step 2: Determine Your OPTIMAL Level of Vitamin DIt’s important to realize the difference between what conventional medicine considers to be “normal,” versus what is optimal. The “normal” 2. 5- hydroxyvitamin D lab range is between 2. As you can see in the chart below, this conventional range is really a sign of deficiency, and is too broad to be ideal. In fact, your vitamin D level should never be below 3. The OPTIMAL value that you’re looking for is 5. This range applies for everyone; children, adolescents, adults, and seniors. These ranges are based on healthy people in tropical or subtropical parts of the world, where they are receiving healthy sun exposures. It seems more than reasonable to assume that these values are in fact reflective of an optimal human requirement. It’s worth to clarify here that ng/ml are U. S. Much of the world uses nmol/l. If your test results are measured in nmol/l, simply multiply the above values by 2. Keeping your level in this range, and even erring toward the higher numbers in this range, is going to give you the most protective benefit. And the way you maintain your levels within this range is by getting tested regularly – say two to four times a year in the beginning, and adjusting your vitamin D intake accordingly. Are Oral Vitamin D Supplements Your Best Choice? The best way to optimize your vitamin D levels is through appropriate safe sunshine or safe tanning bed exposure. However, there are many times when it can be nearly impossible to get enough sun. The darker your skin is, the farther away from the equator you are, and the further away you are from the summer months, the less likely it is that you will produce adequate vitamin D levels from sun exposure alone. In these cases, supplementing with vitamin D is acceptable, but I strongly recommend you monitoring your blood levels regularly when taking oral vitamin D supplements to make sure you’re staying within the optimal range. Only Supplement with the Right Kind of Vitamin DThere is one other thing you need to be aware of if you choose to use an oral vitamin D supplement and that is that there are basically two types – one is natural and one is synthetic. The natural one is D3 (cholecalciferol), which is the same vitamin D your body makes when exposed to sunshine The synthetic one is vitamin D2, which is sometimes called ergocalciferol Once either form of the vitamin is in your body, it must be converted to a more active form. Vitamin D3 is converted 5. D2, and is clearly a better alternative. Vitamin D2 also has a shorter shelf life, and its metabolites bind with protein poorly, making it less effective. Studies have even concluded that vitamin D2 should no longer be regarded as a nutrient appropriate for supplementation or fortification of foods (although it continues to be used). So if you choose to use vitamin D supplements, make sure it is in the form of vitamin D3. Please be aware that nearly all the prescription- based supplements contain synthetic vitamin D2, so if you receive a prescription for vitamin D from your doctor, you’re most likely receiving the inferior vitamin D2. Getting the Word Out About the Benefits of Optimizing Vitamin D Levels. When it comes to the benefits of optimizing your vitamin D levels, the evidence is simply overwhelming. Research shows you can drastically reduce your risk of cancer and countless other chronic diseases by getting safe sun exposure, using a safe tanning bed, or taking a high- quality supplement. Yet, a great deal of people around the world have heard nothing of this great “discovery.” It’s even likely that your doctor is among them, which is why it’s so important to educate yourself. As a result of flawed assumptions about sun exposure, and the subsequent recommendations, a vast majority of people are deficient in vitamin D. It’s thought that over 9. U. S. Plus, there’s no money to be made on selling vitamin D (it’s one of the most inexpensive supplements around) and sun exposure is free! So don’t count on any major corporations or drug companies to help get the message out (rather, count on them to try and suppress this lifesaving information). The longer this information goes largely unnoticed, the more people who will die unnecessarily from potentially preventable cancers and other diseases. Fortunately, Grassroots. Health D*Action is on a mission to get the word out and solve the vitamin D deficiency epidemic. Garland at the helm, The D*Action Project will be monitoring, for five years, the health outcomes of individuals who get their vitamin D levels to the levels of 4. I would highly recommend that you optimize your levels to the high end of this spectrum, as optimal vitamin D levels are 5. Says Carole Baggerly, director of Grassroots. Health. With the expansion of the project by individuals, we could substantially reduce this epidemic in a few years!”So how can you get involved? Join the D*action Project! Simply complete a health questionnaire and test your vitamin D levels two times per year during the five- year program to help demonstrate the public health impact of this nutrient. Grassroots. Health is sponsoring the use of blood spot test kits (laboratory analysis done by ZRT Labs) for a $4. The tests are to be done twice a year by each individual along with the submission of some basic health data. The fee includes: A vitamin D blood spot test kit to be used at home (except in the state of New York) The results are sent directly to you You will be asked to take a quick health survey and also to take action to adjust your vitamin D levels to get into the desired ranges, ideally in consultation with a knowledgeable health care professional. Vitamin D - Wikipedia. Vitamin D refers to a group of fat- soluble secosteroids responsible for increasing intestinal absorption of calcium, iron, magnesium, phosphate, and zinc. In humans, the most important compounds in this group are vitamin D3 (also known as cholecalciferol) and vitamin D2 (ergocalciferol). Vitamin D is made in the skin from cholesterol dependent on sun exposure (specifically UVB radiation). Vitamin D from the diet or dermal synthesis from sunlight is biologically inactive; activation requires enzymatic conversion (hydroxylation) in the liver and kidney. Evidence indicates the synthesis of vitamin D from sun exposure is regulated by a negative feedback loop that prevents toxicity, but because of uncertainty about the cancer risk from sunlight, no recommendations are issued by the Institute of Medicine (US) for the amount of sun exposure required to reach vitamin D requirements. Accordingly, the Dietary Reference Intake for vitamin D assumes no synthesis occurs and all of a person's vitamin D is from food intake. As vitamin D is synthesized in adequate amounts by most mammals exposed to sunlight. Its discovery was due to effort to find the dietary substance lacking in rickets (the childhood form of osteomalacia). Ergocalciferol (vitamin D2) is converted in the liver to 2. D2 . These two specific vitamin D metabolites are measured in serum to determine a person's vitamin D status. Calcitriol also affects neuromuscular and immune function. The two major forms are vitamin D2 or ergocalciferol, and vitamin D3 or cholecalciferol; vitamin D without a subscript refers to either D2 or D3 or both. These are known collectively as calciferol. In 1. 93. 5, the chemical structure of vitamin D3 was established and proven to result from the ultraviolet irradiation of 7- dehydrocholesterol. In the developed world, this is a rare disease. This condition is characterized by bow legs. In sunny countries such as Nigeria, South Africa, and Bangladesh, where the disease occurs among older toddlers and children, it has been attributed to low dietary calcium intakes, which are characteristic of cereal- based diets with limited access to dairy products. Characteristics of this disease are softening of the bones, leading to bending of the spine, bowing of the legs, proximal muscle weakness, bone fragility, and increased risk for fractures. Osteomalacia is usually present when 2. D levels are less than about 1. L. Black women have an increase in serum parathyroid hormone at a lower 2. OH)D level than white women. The threshold for vitamin D toxicity has not been established; however, according to some research, the tolerable upper intake level (UL) is 4,0. IU/day for ages 9. The FDA advised manufacturers of liquid vitamin D supplements that droppers accompanying these products should be clearly and accurately marked for 4. IU). In addition, for products intended for infants, the FDA recommends the dropper hold no more than 4. IU. One thousand micrograms per day in infants has produced toxicity within one month. If hypercalcemia is not treated, it results in excess deposits of calcium in soft tissues and organs such as the kidneys, liver, and heart, resulting in pain and organ damage. Kidney damage may be irreversible. Exposure to sunlight for extended periods of time does not normally cause vitamin D toxicity. The concentrations of vitamin D precursors produced in the skin reach an equilibrium, and any further vitamin D produced is degraded. The review concluded that ranges from 7. L were to be recommended for athletes. Other countries often use nmol/l; ng x 2. In 2. 01. 1 an IOM committee concluded a serum 2. D level of 2. 0 ng/ml (5. The dietary reference intakes for vitamin D are chosen with a margin of safety and 'overshoot' the targeted serum value to ensure the specified levels of intake achieve the desired serum 2. D levels in almost all persons. No contributions to serum 2. D level are assumed from sun exposure and the recommendations are fully applicable to people with dark skin or negligible exposure to sunlight. Serum 2. 5- hydroxyvitamin D levels above 5. Research on vitamin D supplements, including large- scale clinical trials, is continuing. Use of vitamin D in children with normal vitamin D levels does not appear to improve bone density. As of August 2. 01. The greatest benefit with supplementation is seen in athletes who are deficient (2. OH)D serum levels < 3. OH)D serum levels < 2. Incremental decreases in risks are observed with rising serum 2. OH)D concentrations plateauing at 5. Reviews have described the evidence as being . However, lower vitamin D concentrations is also associated with poor nutrition and spending less time outdoors. Therefore, alternative explanations for the increase in cognitive impairment exist and hence a direct causal relationship between vitamin D levels and cognition could not be established. Circulating calcifediol may then be converted into calcitriol, the biologically active form of vitamin D, in the kidneys. Following the final converting step in the kidney, calcitriol is released into the circulation. By binding to vitamin D- binding protein, a carrier protein in the plasma, calcitriol is transported to various target organs. When synthesized by monocyte- macrophages, calcitriol acts locally as a cytokine, defending the body against microbial invaders by stimulating the innate immune system. This reaction is catalyzed by the microsomal enzyme vitamin D 2. Once made, the product is released into the plasma, where it is bound to an . This product is a potent ligand of the vitamin D receptor, which mediates most of the physiological actions of the vitamin. The conversion of calcifediol to calcitriol is catalyzed by the enzyme 2. D3 1- alpha- hydroxylase, the levels of which are increased by parathyroid hormone (and additionally by low calcium or phosphate). Biosynthesis. Second, previtamin D3 spontaneously isomerizes to vitamin D3 (cholecalciferol) in an antarafacialsigmatropic . At room temperature, the transformation of previtamin D3 to vitamin D3 in an organic solvent takes about 1. The conversion of previtamin D3 to vitamin D3 in the skin is about 1. Although primitive vertebrates in the ocean could absorb calcium from the ocean into their skeletons and eat plankton rich in vitamin D, land animals required another way to satisfy their vitamin D requirement for a calcified skeleton without relying on plants. Land vertebrates have been making their own vitamin D for more than 3. Dehydrocholesterol reacts with UVB light at wavelengths between 2. Exposure to light through windows is insufficient because glass almost completely blocks UVB light. The darker the skin, and the weaker the sunlight, the more minutes of exposure are needed. Vitamin D overdose is impossible from UV exposure; the skin reaches an equilibrium where the vitamin degrades as fast as it is created. Vitamin D is produced in the keratinocytes. In birds and fur- bearing mammals, vitamin D is generated from the oily secretions of the skin deposited onto the feathers or fur and is obtained orally during grooming. VDR activation in the intestine, bone, kidney, and parathyroid gland cells leads to the maintenance of calcium and phosphorus levels in the blood (with the assistance of parathyroid hormone and calcitonin) and to the maintenance of bone content. Vitamin D deficiency can result in lower bone mineral density and an increased risk of reduced bone density (osteoporosis) or bone fracture because a lack of vitamin D alters mineral metabolism in the body. Vitamin D also affects the immune system, and VDRs are expressed in several white blood cells, including monocytes and activated T and B cells. British doctor Edward Mellanby noticed dogs that were fed cod liver oil did not develop rickets and concluded vitamin A, or a closely associated factor, could prevent the disease. In 1. 92. 2, Elmer Mc. Collum tested modified cod liver oil in which the vitamin A had been destroyed. He called it vitamin D because it was the fourth vitamin to be named. Alfred Fabian Hess stated: . A meeting took place with J. B. S. Bernal, and Dorothy Crowfoot to discuss possible structures, which contributed to bringing a team together. X- ray crystallography demonstrated the sterol molecules were flat, not as proposed by the German team led by Windaus. In 1. 93. 2, Otto Rosenheim and Harold King published a paper putting forward structures for sterols and bile acids which found immediate acceptance. In the 1. 93. 0s, Windaus clarified further the chemical structure of vitamin D. A vitamin D deficiency is a known cause of rickets. Using $3. 00 of his own money, Steenbock patented his invention. His irradiation technique was used for foodstuffs, most memorably for milk. By the expiration of his patent in 1. US.<. In the liver, vitamin D was found to be converted to calcifediol. Calcifediol is then converted by the kidneys to calcitriol, the biologically active form of vitamin D. The vitamin D metabolites, calcifediol and calcitriol, were identified by competing teams led by Michael F. Holick in the laboratory of Hector De. Luca and by Tony Norman and colleagues. The recommended daily intake of vitamin D may not be sufficient if sunlight exposure is limited. This dose should be increased to 4,0. IU/day in some patients with very low vitamin D status or in case of comorbid conditions. For vitamin D labeling purposes 1. Daily Value was 4. IU (1. 0 . A table of the pre- change adult Daily Values is provided at Reference Daily Intake. Food and supplement companies have until July 2. Upper intake levels. The recommendations were formed assuming the individual has no skin synthesis of vitamin D because of inadequate sun exposure. The reference intake for vitamin D refers to total intake from food, beverages and supplements, is intended for the North American population, and assumes that calcium requirements are being met. Proponents of this view, who include some members of the panel that drafted a now- superseded 1. Hearing Loss appears to be prevented and treated with vitamin DBy getting stronger ear bones. Bought my 9. 8 year- old father- in- law to an audiologist yesterday (Oct 1. I realized that his hearing had improved recently. He has been taking 1. IU of vitamin D and co- factors by Jarrow of Calcium, Magnesium, vitamin K2, etc for the past 9 months. Wondered if there was a correlation. Using Google, I found that hearing- loss . Strikingly, correcting the vitamin D deficiency often corrects the hearing loss and even the deafness in these specific cases. Researchers had to conclude that vitamin D deficiency is likely one of the causes and supplementation should be considered in persons with hearing loss. Vitamin D can be inhibited if there is also a magnesium deficiency. If there is a magnesium deficiency, this will also affect calcium intakes. As you can see, not just one nutrient can be isolated as being a cause or cure of something. Extensive article of Jan 2. Townsend Letter for Doctors, written by Dr. Since then, research findings have been conflicting, some reporting significant vitamin D deficiency, (1- 2) and others not. If you don't have enough vitamin D in your system, this causes insufficient amounts of calcium in the fluid of the inner ear. If you don't have enough calcium, the small bones of the inner ear become spongy. This spongy state interferes with sound transmission. Unless an older person is getting many hours of sunshine daily or taking vitamin D supplements the vitamin D levels are known to greatly decline. The skin produces less than 1/3 as much vitamin D per minute for an older person due to lack of pre- vitamin D in the skin. So the lack of vitamin D which weakens the bones and causes hip fractures, etc also weakens the bones in the ear which are vital for hearing. For example, the 1. National Health Interview Survey (Pleis & Coles, 2. Asian and African decent were less likely to experience hearing problems (7. Whites or Native Americans (1. Whereas 1. 5% of non- Hispanic White adults reported some form of hearing difficulty, 6% of non- Hispanic Black adults and 6% of Hispanic White adults reported hearing problems. Also known as presbyacusis, it is defined as a progressive bilateral symmetrical age- related sensorineural hearing loss. May; 9. 7(5): 4. 05- 2. PMID: 6. 60. 21. 94. Ten patients are reported with bilateral cochlear deafness which was associated with vitamin D deficiency. It should be considered in the differential diagnosis of unexplained bilateral cochlear deafness and may be important in the origin of some cases of otosclerosis, presbyacusis, and the deafness associated with chronic renal failure. Treatment should prevent progressive hearing loss, which may occasionally be partly reversible, and the development of clinical osteomalacia with more generalized skeletal symptoms. Jun; 9. 3(3): 3. 13- 2. The population comprised 2. The disease was bilateral in 4. The mean duration of symptoms was 1. Vitamin D status was evaluated by measuring the plasma 2. D3 (2. 5- OHD), which is the main storage metabolite. Abnormally low 2. OHD levels were found in 1. Raised serum alkaline phosphatase levels were present in 3. Calcium and vitamin D replacement therapy resulted in significant hearing improvement in 3 of 1. Vitamin D deficiency is probably a factor in the etiology of some cases of otosclerosis and is important, since the deafness resulting from cochlear involvement may be reversible. DNA fragmentation and ultimately Ca*2- mcdiated cell death (5. Dietary factors, such as vitamin D deficiency, have been associated with cochlear dysfunction and disruption in calcium homeostasis in the cochlea of rats (4. Meniere's disease, early progressive sensorineural hearing loss, senile deafness, noise exposure) (5. Examination of populations with metabolic disorders associated with impaired calcium and/or vitamin D status (metabolic bone disease, hypoparathyroidism or abnormal blood indices of calcium, phosphate, or alkaline phosphatase) (5. Assessment of vitamin D status in case- control studies comparing hearing impaired vs normal hearing older adults (2. Limitations in the animals studies included incomplete information on dietary formulation and composition; strong possibility that control and experimental diets differed in composition and were lacking in other essential nutrients; limited information of food intake, body weight, and body weight gain; and lack of biochemical confirmation of nutrient deficiencies. Also, one of the earliest studies on nutritional deficiencies and hearing loss in animals was published in 1. These researchers concluded that vitamin D deficiency is likely one of the causal factors in some cases of otosclerosis, cochlear deafness, and presbycusis. According to Brookes (1. If You Are Heavy, You Need More Vitamin DBy Dr. Mercola. Vitamin D is a fat- soluble, hormone- like vitamin, and many therefore believe that if you're obese you need more of it because body fat acts as a . This was not previously widely appreciated by most experts. Your Body Weight May Dictate How Much Vitamin D You Need. If you're overweight or obese, you're therefore likely going to need more vitamin D than a slimmer person - - and the same holds true for people with higher body weights due to muscle mass. Your best source for this vitamin is daily exposure to the sun, without sunblock on your skin, until your skin turns the lightest shade of pink. While this isn't always possible due to the change of the seasons and your geographic location (and your skin color), this is the ideal to aim for as it will optimize your vitamin D levels naturally. To use the sun to maximize your vitamin D production and minimize your risk of skin damage, the middle of the day (roughly between 1. During this UVB- intense period you will need the shortest sun exposure time to produce the most vitamin D. If sun exposure is not an option, using a safe tanning bed (one with electronic ballasts rather than magnetic ballasts, to avoid unnecessary exposure to EMF fields) is the next best option. Safe tanning beds have far less of the dangerous UVA than sunlight, while unsafe ones have more UVA than sunlight. If neither of these are feasible options, then you should take an oral vitamin D3 supplement – and this is where the dosage becomes of crucial importance. What's the Correct Dose of Vitamin D? Download Interview Transcript. Based on research published by Grassroots. Health from the D*Action study, the average adult needs to take 8,0. IU's of vitamin D per day in order to elevate his or her levels above 4. Ideally, you'll want your levels to be between 5. As Carole Baggerly, director and founder of Grassroots. Health, noted. We have about 1. IU a day and they're not reaching a potential toxicity level of 2. The study reported data on about over 3,5. It is curvilinear and it flattens, which is why it's even hard to get toxic with a supplement. So if you take oral vitamin D, ideally you should take vitamin K2 as well or use organic fermented foods that are high in vitamin K2, as you need about 1. That said, 8,0. 00 IU is only a ballpark estimate of what most people likely need - - it is simply impossible to make a general recommendation that will cover everyone's needs. For instance, the lighter your skin, the more vitamin D you will produce from sun exposure, and the closer you live to the equator, the more vitamin D the sun on your skin will produce. Similarly, the more you weigh, the more vitamin D you need. So it is an inexact science trying to figure out how much vitamin D your body is able to produce naturally and balancing that with how much you need in supplement form. You really need to be your own vitamin D level sleuth, and I strongly suggest you do the necessary work, because this is truly one of the most powerful vitamins available for your health, and one that the majority of people currently are deficient in. The only way to truly optimize your own vitamin D levels is to work with your doctor, take the 2. OH D blood test and then get sun exposure and/or supplement with a dose somewhere in the range of 5,0. IU, retesting your blood levels after a few months of supplementation. For children below the age of 5 I recommend 3. IU per pound per day and for pregnant women I recommend anywhere from 5,0. IU per day. But again, there really is no . Vitamin D levels tend to be low in obese individuals as well as in those who are physically inactive,i and one researcher even went so far as to state that vitamin D deficiency is the cause of common obesity and metabolic syndrome: ii. Larger body size confers a survival advantage in the cold ambient temperatures and food scarcity of the winter climate by reducing surface area to volume ratio and by providing an energy store in the form of fat mass. In addition, it is suggested that the phenotypic metabolic and physiological changes observed as the metabolic syndrome, including hypertension and insulin resistance, could result from a winter metabolism which increases thermogenic capacity. Common obesity and the metabolic syndrome may therefore result from an anomalous adaptive winter response. The stimulus for the winter response is proposed to be a fall in vitamin D. Why is Vitamin D So Important, Anyway? We've addressed ways to optimize your vitamin D levels . Many people think that vitamin D is really a vitamin, but in reality, the active form of vitamin D is one of the most potent hormones in your body, and regulates more genes and bodily functions than any other hormone yet discovered. Vitamin D is produced as a pro- hormone in your skin after sunlight exposure, and is then converted to the potent hormone form. Without adequate levels of this hormone, you could die, and indeed, many do die from vitamin D deficiency- related causes. Vitamin D could rightly be described as a . Furthermore, when your organs convert the vitamin D in your bloodstream into calcitriol, which is the hormonal or activated version of vitamin D, they can then use it to repair cellular damage, including damage associated with cancer cells and tumors. Theories linking vitamin D to certain cancers have been tested and confirmed in more than 2. Cedric Garland, Dr. PH, professor of family and preventive medicine at the UC San Diego School of Medicine. Other research has linked vitamin D to a number of chronic and acute health conditions including heart disease, infertility, influenza, colds, respiratory tract infections, depression and more.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. Archives
November 2017
Categories |